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Acetylsalicylic acid may also cause gastric irritation.

A plasma salicylate concentration of 300 to 500 mg/L at 6 hour post-ingestion indicates a mild toxicity, 500 to 800 mg/L moderate toxicity and >800 mg/L is considered as severe SA intoxication.

He also synthesized Aspirin because his father, who had severe arthritis, could not tolerate the salicylic acid he was taking for pain relief.
The Kolbe Synthesis for the production of salicylic acid was created by the German chemist Hermann Kolbe.

The mixture was heated on a hot plate until all the salicylic acid was dissolved.

High unbound fraction of salicylate in plasma during intoxication.

Cases of salicylic acid dermal absorption which induced intoxication particularly in children have also been reported (Davies et al., 1979; Clark & Wilson, 1981).

Estimation of serial plasma salicylate poisoning following overdosage in adults revealed that the peak concentration may be delayed up to 24 hours post ingestion (Balali-Mood, 1981).

Acute acetylsalicylic acid poisoning.

Although the clinical value of screening for salicylates in acute poisoning was emphasized (Chan et al., 1995), determination of plasma salicylate concentration is required to confirm the diagnosis and to estimate the severity of salicylate intoxication.

Chronic salicylate poisoning particularly those with metabolic acidosis, hypoglycaemia, lethargy, coma and fits in malaria endemic areas may mimic severe malaria as was investigated in Kenya (English et al., 1996).

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Protein binding of salicylate in uremic and normal plasma.


Salicylate inhibition of antiplatelet effect of aspirin.

Salicylic acid has more irritant effects on gastric mucosa than acetylsalicylic acid and thus has been withdrawn from oral administration for many years.

Percutaneous salicylic acid intoxication.

Ferric Chloride Test for Purity Chemical Reagents: .1M HCl, standardized
.1M NaOH, standardized
95% ethanol
Phenolphthalein solution Step 1 Approximately .5g of the synthesized aspirin was weighed and placed into a 250mL Erlenmeyer flask.

Acidaemia and salicylate poisoning in adults.

Cigarette smoking, chronic salicylate ingestion, metabolic acidosis and the presence of neurological symptoms and signs on admission are strong risk factors for the subsequent development of pulmonary oedema.

Salicylic acid in: Martindale The Extra Pharmacopoeia, 31st Edition.

Application of teething gels containing salicylic acid induced intoxication (Paynter & Alexandre, 1979). 9.2.4 Eye contact 9.2.5 Parenteral exposure 9.2.6 Other

Disturbances of acid-base equilibrium in salicylate intoxication.

Life threatening salicylate poisoning caused by percutaneous absorption of salicylic acid (10% ointment) in a 7-year-old boy with ichthyosis vulgaris was reported (German et al., 1996).

Overcoming Organic Chemistry: Synthesis of Salicylic Acid

The presenting signs of chronic salicylate poisoning can include metabolic acidosis, hypoglycemia, lethargy, coma and fits (English et al., 1996). 9.2.2 Inhalation 9.2.3 Skin exposure

Salicylic acid (from Latin salix, willow tree) ..

of HCL: .0932M
Sample Weight: .516g Volume of NaOH required to neutralize all acids present: Final buret reading: 45.50mL
Initial buret reading: 0mL
Volume of NaOH: 30.50mL
Approximate volume of NaOH to be added for hydrolysis: 45.50mL Actual volume of NaOH added for hydrolysis: Final buret reading: 15.4mL
Initial buret reading: 0mL
Volume of NaOH: 45.50mL 7 6 3 4 6 3 3 4 2 2 3 #1 #2 #3 8 4 4 2 2 Procedure 2 Explanation of Calculations (l) (aq) (aq) (aq) 0.0932 M For all three trials, we used the equation:
M V = M V
Then we rearranged the equation to solve for M by dividing by V .

Biosynthesis of salicylic acid in ..

In patients with significant acidosis and in patients who ingest multiple doses or sustained release preparations, the Done nomogram will tend to underestimate the severity of salicylate intoxication (Todd et al., 1981).

Synthesis of Salicylic Acid - Sciencemadness Wiki

Acid-base disturbances are usually mixed in mild to moderate salicylate poisoning (Proudfoot & Brown, 1969; Proudfoot, 1983). 8.3.3 Haematological analyses 8.3.4 Interpretation of biomedical investigations

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